“Dr. Shankara Chetty of South Africa has closely monitored and treated over 8000 patients personally. He noticed there was something very different about viral pneumonia in comparison to C-V pneumonia and was determined to figure out what the mechanism of treatment should be. He had all of his patients come in as soon as they expressed any symptoms, he followed the progression of everyone’s disease pattern and noticed something very interesting which resulted in his findings.
Every person progressed the same for the first 5 days, all of them, even those who eventually went on to worsen, appeared to improve around that 5th to 6th day. But from that day forward, there were two distinct camps…one group would always go on to improve after that initial period, but the other group would suddenly worsen on day 8. Not day 7 or 9, always day 8, and they would be fine day 7, while going on to develop what we’ve come to recognize as C-V pneumonia. But it’s not pneumonia at all!
Dr. Chetty knew the pattern of expression for pneumonia of all kinds and this C-V “pneumonia” wasn’t behaving like any other form of pneumonia. In fact, the flow of oxygen wasn’t restricted at all, like in pneumonia. Patients were showing a restriction to lung expansion, but not air flow and it was coming on rapidly, unlike pneumonia. The lung itself was inflamed and couldn’t expand.
It was then that he recognized the similarity this condition had with an allergic reaction. The second an allergic reaction happens, inflammation creates instant symptoms. In peanut allergies for example, you eat a peanut and your face swells up. Pneumonia is progressive, not instant like this.
As it turns out, this illness is biphasic, meaning there could potentially be two phases of illness and it’s the second phase where mortality and morbidity occur.
He had a critically ill patient, who had diabetes, hypertension and her oxygen saturation was in the 80’s, both pre-existing conditions are co-morbidities of C-V. He treated her with a high dose steroid and added in an antihistamine.
When he called to check on her the next day, her oxygen levels were normal and she progressed on to full health.
It’s important to note here too that none of his patients have experienced complications of C o V I D or long haulers. He has since begun working with long hauler’s cases though and even in their testing, he is discovering that it’s related to mast cell degranulation – aka – histamine!! . He may be the ONLY doctor tracking this illness so closely. Although, I have noticed that the I-MASK protocols have been altered to include Singulair.
“C o V I D pneumonia is actually mast cell degranulation of the lungs!! Put simply, it’s an allergic reaction occurring after the viral phase ends, most likely to something in the viral particles left over after the body deals with the virus. The reason you can’t tell it’s an allergic reaction is because it’s happening in the lungs, so you experience only symptoms of chest tightness and fatigue!”
I didn’t do the corticosteroid because they gave that too me in the hospital.
Antihistamine – I took benedryl but plan to get some daytime non drowsy antihistamine.
Anti-leukotrienes – I took singulair
Blood thinner – I took aspirin
I also took ivermectin but I don’t think you have to if you can’t get any. 12mg/day for five days.
This is what I found online…
8th day Protocol from Dr Chetty
Intervention #1:
Corticosteroids
Goal: To stop the hypersensitivity reaction, to stop the release of mediators and to prevent an inappropriate immune response, including a possible subsequent cytokine storm.
Medication:
Prednisone 80mg dly x 1 week.
Note: Increase dose rapidly to get symptomatic relief quickly. CRP and IL6 values must show quick decline. Dose will vary according to variants and severity of reaction. Can go as high as 100mg tds for first few days. Wean off cautiously when CRP and IL6 are normal or patient is well for a few days. Those with prolonged reactions are difficult to wean, so consider adding Azathioprine 50mg dly to decrease steroid requirements.
Intervention #2: Anti-histamines
Goal: To clear the histamines that have been released.
Medications:
H1: Promethazine 25mg tds x 5 days
or
Levocetirizine 5mg bd x 1 month to follow Promethazine
H2: Cimetidine 400mg x 1 month or,
another H2 blocker
Other anti-histamine drugs can be suitable
Intervention #3: Anti-leukotrienes
Goal: To clear the leukotrienes that have been released. Medication: Montelukast 10mg bd x 5 days then dly x 1 month
Add Xarelto 15 mg bd if D.Dimer is raised; decrease to 15 mg dly x 1 month once D.Dimer is normal
Optional Interventions
Add appropriate antibiotics for those with fever, bacterial co-infection or raised Procalcitonin levels
Add Venteze syrup PRN for those suffering from asthma
Add Ivermectin 12 mg dly x 5 days in those with cough, dyspnea or decreased oxygen saturation
Fluvoxamine may be a suitable drug, yet Dr Chetty has so far no experience with it.
“Dr. Shankara Chetty of South Africa has closely monitored and treated over 8000 patients personally. He noticed there was something very different about viral pneumonia in comparison to C-V pneumonia and was determined to figure out what the mechanism of treatment should be. He had all of his patients come in as soon as they expressed any symptoms, he followed the progression of everyone’s disease pattern and noticed something very interesting which resulted in his findings.
Every person progressed the same for the first 5 days, all of them, even those who eventually went on to worsen, appeared to improve around that 5th to 6th day. But from that day forward, there were two distinct camps…one group would always go on to improve after that initial period, but the other group would suddenly worsen on day 8. Not day 7 or 9, always day 8, and they would be fine day 7, while going on to develop what we’ve come to recognize as C-V pneumonia. But it’s not pneumonia at all!
Dr. Chetty knew the pattern of expression for pneumonia of all kinds and this C-V “pneumonia” wasn’t behaving like any other form of pneumonia. In fact, the flow of oxygen wasn’t restricted at all, like in pneumonia. Patients were showing a restriction to lung expansion, but not air flow and it was coming on rapidly, unlike pneumonia. The lung itself was inflamed and couldn’t expand.
It was then that he recognized the similarity this condition had with an allergic reaction. The second an allergic reaction happens, inflammation creates instant symptoms. In peanut allergies for example, you eat a peanut and your face swells up. Pneumonia is progressive, not instant like this.
As it turns out, this illness is biphasic, meaning there could potentially be two phases of illness and it’s the second phase where mortality and morbidity occur.
He had a critically ill patient, who had diabetes, hypertension and her oxygen saturation was in the 80’s, both pre-existing conditions are co-morbidities of C-V. He treated her with a high dose steroid and added in an antihistamine.
When he called to check on her the next day, her oxygen levels were normal and she progressed on to full health.
It’s important to note here too that none of his patients have experienced complications of C o V I D or long haulers. He has since begun working with long hauler’s cases though and even in their testing, he is discovering that it’s related to mast cell degranulation – aka – histamine!! . He may be the ONLY doctor tracking this illness so closely. Although, I have noticed that the I-MASK protocols have been altered to include Singulair.
“C o V I D pneumonia is actually mast cell degranulation of the lungs!! Put simply, it’s an allergic reaction occurring after the viral phase ends, most likely to something in the viral particles left over after the body deals with the virus. The reason you can’t tell it’s an allergic reaction is because it’s happening in the lungs, so you experience only symptoms of chest tightness and fatigue!”
I didn’t do the corticosteroid because they gave that too me in the hospital.
Antihistamine – I took benedryl but plan to get some daytime non drowsy antihistamine.
Anti-leukotrienes – I took singulair
Blood thinner – I took aspirin
I also took ivermectin but I don’t think you have to if you can’t get any. 12mg/day for five days.
This is what I found online…
8th day Protocol from Dr Chetty
Intervention #1:
Corticosteroids
Goal: To stop the hypersensitivity reaction, to stop the release of mediators and to prevent an inappropriate immune response, including a possible subsequent cytokine storm.
Medication:
Prednisone 80mg dly x 1 week.
Note: Increase dose rapidly to get symptomatic relief quickly. CRP and IL6 values must show quick decline. Dose will vary according to variants and severity of reaction. Can go as high as 100mg tds for first few days. Wean off cautiously when CRP and IL6 are normal or patient is well for a few days. Those with prolonged reactions are difficult to wean, so consider adding Azathioprine 50mg dly to decrease steroid requirements.
Intervention #2: Anti-histamines
Goal: To clear the histamines that have been released.
Medications:
H1: Promethazine 25mg tds x 5 days
or
Levocetirizine 5mg bd x 1 month to follow Promethazine
H2: Cimetidine 400mg x 1 month or,
another H2 blocker
Other anti-histamine drugs can be suitable
Intervention #3: Anti-leukotrienes
Goal: To clear the leukotrienes that have been released. Medication: Montelukast 10mg bd x 5 days then dly x 1 month
Intervention #4: Blood Thinners
Goal: to clear platelet activating factors
Medications:
Aspirin 325 mg dly x 1 month.
Add Xarelto 15 mg bd if D.Dimer is raised; decrease to 15 mg dly x 1 month once D.Dimer is normal
Optional Interventions
Add appropriate antibiotics for those with fever, bacterial co-infection or raised Procalcitonin levels
Add Venteze syrup PRN for those suffering from asthma
Add Ivermectin 12 mg dly x 5 days in those with cough, dyspnea or decreased oxygen saturation
Fluvoxamine may be a suitable drug, yet Dr Chetty has so far no experience with it.